This was formulated probably 15 years ago, well before the current surge of books on homosapiens.
The theory is based on fixed 50 cell cycles in all mammals.
Linguists claim that all languages share common features.
Basic unit of language which include phonological, syntactic and semantic units are universally used by all languages in construction of communication skills.
In all cultures children quickly and readily learn the sounds and grammatical forms and use them appropriately from very early stages of development.
There is evidence that this capacity to (transmit and receive messages) is an inherited feature of the human brain.
The power of speech come naturally as the child grows (with it the brain maturation) and need not be taught as it is implied by educationalists.
Coming to the cell cycle story, the sudden increase in brain size probably suggests that the emergence of language may have begun less than million years ago, perhaps less than half a million. The rapidity of evolution over time could account for the doubling of of brain size by addition of one further cell division in the sequence of some 30/40 mitosis that exists between the first cleavage and the cessation of division in the nervous system giving 10 to 100 billion cells altogether (The total of 10'14 cells in an adult after 45 cell cycles).
The total cell cycle in human is around 50 cell cycles altogether.
The selective advantage that bestow on humans far outweighed the number/size of the brain that followed this unexplained episode in evolution.
The acceleration and improvement in communication in late stages of evolution gave rise to 'recording the new found tool as wanting' possibly less than 10,000 years ago, corresponding with the discovery of fire and stone tools.
The doubling of cells would have happened before 20 weeks of gestation with proportionate increase in the skull size and its bones most likely cartilage that would have caused premature delivery.
Extension of the theory to first cell division would have given twin births, that itself would have caused premature delivery to begin with.
Along with these cell divisions there would have been relative feminization of the foetus with disappearance of body hair, due either to lack of testosterone sensitivity or the skin segment overlying the rapidly growing skull surface having hair distributed over it, instead of the skin (neuroectodermal in origin) appendage covering the rest of the body.
Considering that the prematurity at least of 4 weeks, the infant would have prolonged nursing period that would have given added advantage of prolonged learning period.
Assuming that the two infants were chosen to be nursed with other nursing mothers which would have encouraged the early vocalization of sounds that were imprinted as it were in the subsequent generations.
All the changes were somatic in nature not needing any drastic mutations as such except that the cell cycle out of place and phase (coming on early) promoting relatively infantile looking (lost of body hair) infant.
The cell cycle out of phase may have contributed profuse hair on the skull and sparing the skin elsewhere (hairless).
One cell cycle out of phase by chance incidence would have caused premature or early pubertal growth and increased fertility and raring of children earlier, assuming both were female infants (lot of assumptions).
The onward shift and the pattern of cell cycle probably remained constant giving this subpopulation a gift of prolonged infantile nursing period in infancy and enhanced fertility when adults. The feminization of this subpopulation would have coincided with less rugged bone structure of the skull that did not hinder vaginal delivery (prematurity+less rugged bone of the skull).
None of the above can be supported by fossil studies and cell cycle shift can even cause advanced senility as well as premature growth and early puberty, considering that the there are only fixed number of cell cycles numbering 50.
It is well known fact with intrauterine growth retardation that there is a shift of cell cycle with advanced intrauterine maturation and early onset of adult diseases such as hypertension and ishaemic heart disease.
The increase of brain cells from 10 billion to 100 billion increased the intercommunication networks in the brain tremendously facilitating memory, learning capacity and language capabilities.
In summary, doubling of genome occurs rarely and once in every 3 to 5 million years.
The relative absence of similar hominods in evolution could be explained by simple cell cycle shift not needing grand genomic changes to account for our evolution.
We are what we are today not necessarily due to grand scale genomics but due to somatic changes that was brought on by simple cell cycle doubling and shifting its mode.
By the way, the skin colour has no relationship with the above scenarios.
The relationship of one melanocyte to 33 odd keratinocytes is constant.
In other words one melanocyte supplies 33 keratinocytes.
It remained black for very long period and the loss of melananosome melanization that occurred in an unknown time created the present American predicament.
If at all White skin should be considered a genetic insufficiency to withstand radiation injury.
The theory is based on fixed 50 cell cycles in all mammals.
Linguists claim that all languages share common features.
Basic unit of language which include phonological, syntactic and semantic units are universally used by all languages in construction of communication skills.
In all cultures children quickly and readily learn the sounds and grammatical forms and use them appropriately from very early stages of development.
There is evidence that this capacity to (transmit and receive messages) is an inherited feature of the human brain.
The power of speech come naturally as the child grows (with it the brain maturation) and need not be taught as it is implied by educationalists.
Coming to the cell cycle story, the sudden increase in brain size probably suggests that the emergence of language may have begun less than million years ago, perhaps less than half a million. The rapidity of evolution over time could account for the doubling of of brain size by addition of one further cell division in the sequence of some 30/40 mitosis that exists between the first cleavage and the cessation of division in the nervous system giving 10 to 100 billion cells altogether (The total of 10'14 cells in an adult after 45 cell cycles).
The total cell cycle in human is around 50 cell cycles altogether.
The selective advantage that bestow on humans far outweighed the number/size of the brain that followed this unexplained episode in evolution.
The acceleration and improvement in communication in late stages of evolution gave rise to 'recording the new found tool as wanting' possibly less than 10,000 years ago, corresponding with the discovery of fire and stone tools.
The doubling of cells would have happened before 20 weeks of gestation with proportionate increase in the skull size and its bones most likely cartilage that would have caused premature delivery.
Extension of the theory to first cell division would have given twin births, that itself would have caused premature delivery to begin with.
Along with these cell divisions there would have been relative feminization of the foetus with disappearance of body hair, due either to lack of testosterone sensitivity or the skin segment overlying the rapidly growing skull surface having hair distributed over it, instead of the skin (neuroectodermal in origin) appendage covering the rest of the body.
Considering that the prematurity at least of 4 weeks, the infant would have prolonged nursing period that would have given added advantage of prolonged learning period.
Assuming that the two infants were chosen to be nursed with other nursing mothers which would have encouraged the early vocalization of sounds that were imprinted as it were in the subsequent generations.
All the changes were somatic in nature not needing any drastic mutations as such except that the cell cycle out of place and phase (coming on early) promoting relatively infantile looking (lost of body hair) infant.
The cell cycle out of phase may have contributed profuse hair on the skull and sparing the skin elsewhere (hairless).
One cell cycle out of phase by chance incidence would have caused premature or early pubertal growth and increased fertility and raring of children earlier, assuming both were female infants (lot of assumptions).
The onward shift and the pattern of cell cycle probably remained constant giving this subpopulation a gift of prolonged infantile nursing period in infancy and enhanced fertility when adults. The feminization of this subpopulation would have coincided with less rugged bone structure of the skull that did not hinder vaginal delivery (prematurity+less rugged bone of the skull).
None of the above can be supported by fossil studies and cell cycle shift can even cause advanced senility as well as premature growth and early puberty, considering that the there are only fixed number of cell cycles numbering 50.
It is well known fact with intrauterine growth retardation that there is a shift of cell cycle with advanced intrauterine maturation and early onset of adult diseases such as hypertension and ishaemic heart disease.
The increase of brain cells from 10 billion to 100 billion increased the intercommunication networks in the brain tremendously facilitating memory, learning capacity and language capabilities.
In summary, doubling of genome occurs rarely and once in every 3 to 5 million years.
The relative absence of similar hominods in evolution could be explained by simple cell cycle shift not needing grand genomic changes to account for our evolution.
We are what we are today not necessarily due to grand scale genomics but due to somatic changes that was brought on by simple cell cycle doubling and shifting its mode.
By the way, the skin colour has no relationship with the above scenarios.
The relationship of one melanocyte to 33 odd keratinocytes is constant.
In other words one melanocyte supplies 33 keratinocytes.
It remained black for very long period and the loss of melananosome melanization that occurred in an unknown time created the present American predicament.
If at all White skin should be considered a genetic insufficiency to withstand radiation injury.
