Did our Ceylonese Dog solved the problem of the Russian President?

Did our Ceylonese dog solved the problem of the Russian President? 

Unlike on earth Paranoia is absent in heaven. 

Paranoia precludes dementia in humans. 

Maha wants to find the genomic piece of paranoia in these two dogs and transplant the piece into his divine plotters.

It is win win situation.

He gets his competitors wrapped up in delusion of grandiosity. 

I get a scheme to make a fast buck by copyrighting this genomic sequence while having a free ride to Russia. Russia has banned Ceylonese from traveling to Russia unless he or she has some blood relationship to a real Russian. I did so, by offering to solve the dog problem of Russian President Vladimir Putin.

Did our Ceylonese dog solved the problem of the Russian President?

Yes, he did.

How?

It very simple logic.

He was made to delver the food to the dogs.

The plates were kept outside and the Russian dog has to step outside from the kennel and eat. Until he eats our Ceylonese dog does not touch his food. Every day, the plate is kept further away from the his kennel and more towards the American dog’s kennel until they were facing each other.

It is all sign language in practice.

The dog handlers job is to prompt the name of his dog. 

In about three months both Russian dog and the American dogs realized they had a name of their own.          

I suppose our Ceylonese dog should get a Nobel Prize for this feat, since no Ceylonese has being nominated so far. The bonus for me is when the award ceremony is held in Stockholm Sweden, I get a chance to accompany the dog as a handler. 

I have refused to become a dog handler after the demise of our dog Zimba.    I would not say anything about Zimba until, I meet him in heaven. Until then, all my passwords for NUC computer would have some element of his name attached as a souvenir.

Coming to the present predicament, even though the dogs had dementia none of the dogs lost their natural ways. American dog had American habits, Ceylonese Paraya dog had stray dog habits. Only exception was the Russian dog. He had lost the canny ability to learn a foreign language, in 10 days, if left alone in the foreign airport. This was a real asset for the Russian President and now he has lost it. 

He was looking for some remedy.

Our president with my initiation send a message to Russian embassy that we could solve using one of our own dogs. The only caveat was we had to get permission form Chinese authorities. 

Russians did not have any objections.

This how it was accomplished.

A strain of Wuhan virus was the intermediary.

Our Paraya dog has to be taken to China for 10 days and was kept with a Chinese special dog. They gain in function part of the virus is changed to the gene for Chinese language. Within 10 days our Ceylonese dog gain the ability to converse in sign language of Chinese.  Then he was taken to Russia and was kept with the Russian dog. Within 10 days the Russian dog acquires Chinese capability. 

For Russians this was real asset. 

However, much they trust Chinese diplomats having a Russian dog who could see the real intent of Chinese, at least in sign language was an asset that Russia would not have dreamt of.

What about the American dog, who lost his ability in English?

It is simple Ceylonese dog is kept with the American dog for ten day he acquires the typical Ceylonese English of American descent.

I am left with two problems.

I cannot get the Russian dog to comprehend Russian but he is well versed with English.

I cannot get the Ceylonese dog to comprehend English. 

He can comprehend only Sinhala language.

They have lost the parts of the genome of language comprehension which is closely linked to the dementia portion. 

Loss of language ability is part of the syndrome of dementia anyway.

So I have decided to send Ceylonese dogs to heaven, so Maha Brahama Assistant who is a Pedigree dog can use his genomic material to change his rival God’s genes, so that they become demented and won’t be a challenge to his regime in heaven. 

I have no regret letting this Ceylonese dog succumb to a natural death due to food scarcity already evident in Ceylon.

But before that event happening, I have to help President Donald Trump to get my dogs to help in CDC investigation in detail. This would take at least a few years and Junior John F. Kennedy would be happy to accept him as a gift from Ceylon.

NuTyX

NuTyX 

NuTyX is a very flexible, complete GNU / Linux distribution for advanced or motivated users.

Numerous concepts of the distribution are original and therefore exist only on NuTyX.

For example, it has its own CARDS package manager.

You can choose to use the ports system, compile and install your packages from their sources, they will be integrated natively in the system, their management will be transparent.

In addition to the traditional ISOs available, an installation script can install NuTyX from another distribution.
 

Features

    Own CLI/GUI Package Manager
    Simple script to install from another Linux OS
    Testing and Rolling releases
    KDE Plasma 5 Desktop Environment
    GNOME Desktop Environment
    LXQt Desktop Environment
    Budgie Desktop Environment
    Cinnamon Desktop Environment

Mandrake Linux 7.0

Mandrake Linux 7.0

by  MandrakeSoft
Publication date  2000-05

Topics Linux, Linux Mandrake, CD, Operating System

Language  English

Item Size  641.1M

Version 7.0 of Mandrake Linux as included in Maximum Linux, May/June issue of 2000

Added date 2021-12-06 00:21:04

Identifier   mandrake-linux-7.0

Scanner Internet Archive HTML5 Uploader 1.6.4

Year 2000

Mandriva Linux

Mandriva Linux

Web site: mandriva.com/en/linux/ (not active)
Origin: France
Category: Desktop
Desktop environment: GNOME, KDE, LXDE, Xfce
Architecture: x86, x86_64
Based on: Independent, Red Hat
Wikipedia: Mandriva Linux
Media: Live CD/DVD, Install DVD
The last version | Released: 2012 Alpha 2 | November 6, 2012
Zobacz po polsku Zobacz po polsku: Mandriva Linux

Mandriva Linux (previously: Mandrake Linux) – an independent Linux distribution, originally forked from Red Hat Linux.

The system features the most popular desktop environments, a set of pre-installed application for daily usage, and integrated its own graphical configuration utilities tool ‘Mandriva Control Center’.

Mandriva Linux uses ‘urpmi’ package manager, which allows installation of packages by automatically installing all the needed dependencies, from various media, including network/Internet, CD/DVD and local disk. Urpmi has a graphical front-end called ‘pmdrake’, which is integrated into the Mandriva Control Center.
The RPM package management system is also available in the system to perform operation on the packages.

Mandriva Linux was started in 1998 under the name of Mandrake Linux.
The first release was based on Red Hat Linux 5.1, used the K Desktop Environment 1, and was released in July 1998.
In February 2005, MandrakeSoft merged with Brazil’s company called Conectiva to the new Mandriva S.A. In May 2015 Mandriva SA has been closed.

The last stable version of Mandriva Linux 2011 was released in August 2011.

Derivatives:
– Most Mandriva Linux developers went to Mandriva fork called Mageia.
– ROSA is a fork of Mandriva created by former Mandriva developers too.
– OpenMandriva Lx has been built on the top of ROSA as its fork.
– PCLinuxOS initially derived from Mandrake.
– Unity Linux (discontinued) is a Mandriva based distribution.

DemoLinux

DemoLinux

Web site: www.demolinux.org
Origin: France
Category: Desktop
Desktop environment: Gnome, KDE
Architecture: x86
Based on: Debian
Wikipedia: DemoLinux
Media: Live CD
The last version | Released: 3.0.1 | January 20, 2002

DemoLinux – one of the first Live CD Linux distributions, and was created to make it possible to use Linux without having to install it on the hard disk. 

It is the first Linux Live CD making possible to use the system in graphic mode and without any stage of configuration.

Version 1 was based on Mandrake Linux (now Mandriva Linux), versions 2 and 3 used a mechanism independent of the distribution and were distributed mainly on a Debian basis.

Version 3.0 now introduces the Xvesa X server, replacing the framebuffer used before, which depended on VESA 2.0 cards. We now handle PCI sound cards, Lucent winmodems, Reiserfs version 3.5.x, certain USB peripherals and several other 2.2.18 kernel devices. As for version 2.0, DemoLinux 3 heavily uses a transparent compression schema that allows to store (sotre) over a gigabyte of applications, including GNOME and KDE and the StarOffice office suite.

To try out DemoLinux, you need a PC equipped with a CD-ROM and at least 32 MB of RAM for simple graphics interfaces, 64 for advanced graphics interfaces, 128 to use StarOffice.

DemoLinux project was composed of 3 people, all based at the Paris VII University of Paris: 

Vincent Balat, 

Roberto Di Cosmo and 

Jean-Vincent Loddo.

The project was under development between 2000 and 2002.

Demo Linux and the Nostalgia

September 23, 2011

Demo Linux and the Nostalgia
It was a very pleasant experience to boot the Demo Linux that I downloaded 2 days ago.
It did not boot up with my ThinkCentre but did boot up with the NetVista without a problem an even detected the USB ports and the paraphernalia attached to the computer.
It had KDE, Gnome and surprisingly StarOffice 2 (which I had never used before).
Most of the utilities were Debian based and it did not have office package not even AbiWord but there were 10 or more text editors including LyX, Latex derivative and Quanta 2 the web editor too.
 

There were hundred of utilities and games and demo did not need a password to boot.
It was very quick to boot and had all the text stating what was happenings behind the scene.
With tablets coming to scene, the humble beginning of Linux and now almost 95% of utilities including Androids using Linux is an amazing feat of the last 20 years.
But thank goes to Live CD List web site for keeping an archive CD there for downloading.
Now I am going to download version 2.
I used version 3 for testing.
Now that distromania is defunct I hope Live CD List keep some copies of old Linux for the future generation to see as a historical fact.

Without history mankind does not exist even in the digital age!

It reminded me of the Live Suse CD I used before I migrated from Redhat and Debian to Suse.

Leonard Hayflick and 50 Cell Cycle

 Reproduction

Leonard Hayflick and 50 Cell Cycle 

LimitEponyms have long been a feature of medicine and science. The textbooks are dotted with eponymous syndromes and diseases, with laws and constants, all flattering the memory of their inventors or discoverers. But there is only one eponymous limit in biomedicine: the Hayflick Limit, the number of times (about 50) that normal human embryonic cells can divide before they succumb to senescence.Leonard Hayflick, professor of anatomy at the University of California at San Francisco, advanced the concept 50 years ago. The Hayflick Limit, he contended, was both an explanation for the phenomenon of ageing and a demolition of the wishful view (of some) that the human lifespan need have no upper limit. But although he correctly identified the cell nucleus as the location of the responsible mechanism, it fell to others to discern the structures involved. It was biologists Nobel Prize winners Elizabeth Blackburn and Carol Greider who showed how the cell keeps a tally of the number of times it has divided during its progress towards the Hayflick Limit. The ends of chromosomes carry structures called telomeres. Every time a cell divides the telomeres become shorter, this loss being the basis of what Hayflick described not as a clock (the process is not dependent on measuring time) but as a counting device, a “replicometer”.

Now 83 years old, Hayflick no longer has his own laboratory, but is still closely involved with ageing. He writes, lectures, does consultancy work, and, in a satisfying manifestation of natural justice, reckons that the ageing process is treating him well. “I think I'm pretty much ahead of the game for two reasons”, he says. 

“First of all I've absolutely no chronic medical problems that I'm aware of. And, perhaps more important, my mother is about to celebrate her 105th birthday.” 

The genes, if not the telomeres, are on his side.

Life expectancy in most developed countries is, and has for many years, been creeping upwards: some 25 years over the past century. Much of this is the consequence of success at dealing with our traditional killer, infectious diseases. 

But the rate of increase, Hayflick contends, will diminish as we approach what he believes to be our natural average lifespan of around 92 years. 

America's thriving anti-ageing movement annoys him greatly. “The invention of ways to increase human longevity is the world's second oldest profession, or maybe even the first. Individuals are going to the bank at this moment with enormous sums of money gained by persuading people that they've found either a way to extend your life or to make you immortal.” To imagine that the current rate of life expectancy increase will continue indefinitely is as absurd as extrapolating the diminishing time taken to run a mile and concluding that it will sooner or later be done in one second. “Everything in the Universe changes or ages with time, and to think that you can reverse it is nonsense.” When asked how he responds to the idea in principle of finding some way to postpone death indefinitely, he responds with dry humour that any such development would be expensive, and therefore available only to the rich and powerful. “I don't know how many rich and powerful friends you have, but some of the ones I have I certainly don't want to live another decade or two beyond normal.”

Hayflick's career has not been without struggle. That he looks back with no regrets surely reflects that he emerged from his two major disputes so triumphantly. The first concerned the discovery that bears his name. Ageing research generated little interest in the USA when he first entered it. It had been dogma for 60 years that the eventual death of normal human cells in culture was not due to some inherent property, but caused by ignorance of the proper conditions under which to culture them. It took what Hayflick describes as “10 or 15 painful years” for the scientific community to accept what he'd discovered. “To torpedo a half century old belief is not easy even in science.”

The other and potentially more damaging dispute was with the US Government. 

 The conflict began in the early 1960s when Hayflick was working at the University of Pennsylvania's Wistar Institute. 

He developed a cell line dubbed WI-38. 

Because it was living material it couldn't then be patented, so the vaccine companies to which it was freely distributed (it had proved to be exceptionally good for growing viruses) were earning a commercial return while the institution and the individuals who had created it were not. 

Hayflick set up his own company to distribute WI-38 and to hold payments for packaging and shipping until ownership was settled. 

The National Institutes of Health (NIH), which had previously supported the distribution, then accused Hayflick of stealing government property. 

He in turn filed a lawsuit against the NIH. 

After 6 years the action was settled out of court on terms that allowed Hayflick to continue distributing WI-38. 

The subsequent passage of the 1980 Bayh-Dole Act specifically allowed researchers at universities to apply for patents on federally funded inventions.

“What I had done was embraced as policy by the US Government”, says Hayflick, who seems to look back on the saga less in sorrow than in glee. 

“Had my ideas not prevailed there would be no biotechnology industry.” 

It was, after all, mostly scientists who'd developed new techniques and materials while supported by public funds who went on to set up the small companies which created that industry. “I consider it one of my most important achievements.” 

As a researcher, Hayflick has shaped form as well as content.

Dementia Update

 

Dementia Update

I do not think dementia is an invariable accompaniment of old age but part of the Total End Organ Failure that is determined by Cell Cycle Events and the inability to Heal or Repair, lost cells.

Healing and Repair are inherent processes which we are born with. It cannot be upgraded.

One is free to share this with kith and kin.

This piece is in development stage. If one modifies the contents one is responsible (copyright) for his or her own vagaries. I am a scientific man and nothing is granted as real or fully understood. Do not take any concept as acceptable until proven otherwise. 

Just apply cause and effect principle as the driving force with Kalama Sutta as the safeguard for any diversion in focus of attention. 

Let me take the putative causes first.

1. Lack of proper sleep. It is in reality disturbance of diurnal melatonin secretion.

Early to rise and early to bed is the bottom line.

2. Alcohol consumption on regular basis as a energizing tonic.

Alcohol is a universal cell poison including neurons.

3. Over use of cellphone that effect on and off switch of brain frequencies. It is much complicated than that. The new term plasticity of brain is a misnomer. Living under giant Telecom Towers even affect grassing cattle. Computers and television screens are included in this frequency modulation effect.

4. Too much attention to Meditation and physical inactivity. It means self imposed lack of physical exercise.

Walking meditation is probably better than sitting meditation.

5. Fifth is most abundant and damaging.      Strict conformity to a bizarre religious dogmatism that can cause Third World War.

Currently it is in active mode.

6. Some medicinal derivatives, especially cholesterol lowering drugs. Drug companies are hiding these facts and I have documented this elsewhere in detail.

7.Ayahuasca consumption with Spiritual Upliftment (in fact a grand delusion). It is a drug. Drug induced delusional states are as worse as dementia.

8. Consumption of beef.

This is related to Amyloid Proteins which are not broken down to amino acids and deposited in brain in nano quantities to begin with. Their ongoing assimilation to bigger molecules is automatic and do not need catalytic activity of enzymes.

9. Certain viruses cause brain damage. Savages in Papua New Guinea known for cannibalism who eat brains of the victims harbour  these viruses. Kuru, a prion disease, is the disease that affected the people of Papua New Guinea. It is not a virus, but an infectious protein. Mad cow disease, or bovine spongiform encephalopathy (BSE), is a disease that was first found in cattle. It is related to a disease in humans called variant Creutzfeldt-Jakob disease. It is directly related to eating beef of cattle that suffered from BSE. This virus was generally named as Mad Cow Disease Virus. Mrs. Thatcher in 1980s as the PM in UK suppressed this Scientific Information deliberately, to safeguard British beef industry. I was in UK during this period and left UK never to return.

Ready made Remedies

1. Avoid above putative causes and have 8 or 9 hour sleep rhythm. Power snooze (nap) of one (1) hour on the desk is mandatory at least for university students and school children.

In practice 4 hours of Deep Sleep is enough and Lord Buddha was a good exponent of this strategy and he used his waking hours to the benefit of mankind.

By the way, he walked length and breath of India.

Sleep less and work more for your fellow beings. Do not work like Ceylonese Civil Servants. They take a big lunch and maintain a prolong absence from the desk as if they are busy with some urgent work.

Better skip lunch.

Memory which I have not discussed here is established in these 4 hours of Deep Sleep.

Breaking Sleep and overnight cramming is bad.  I never had this habit and slept well the day before the examination. It was bit difficult at first but one has to cultivate it gradually.

2. Use plenty of Pol Sambol (kernel) and coconut oil for food. Coconut fat has medium change fatty acids and are readily metabolized. Coconut does not contribute to obesity. Besides, the ketone bodies provide ready made energy even for dementia patients.

It is a fact.

Coconut water (has an excess of potassium) is iso-osmotic and provides electrolytes in a suitable package.

3. Missing one meal out of three (single good meal strategy) with a good breakfast. Hunter gatherer survived on one meal.

A good banana and a good cup of Ceylon Tea (not coffee) for breakfast are adequate.

Too much Tea effects your sleep.

4. Regular Exercise at least twice a week.

5. Lots of fruits and vegetables with antioxidants including cashew nuts. Cashew is a mild aphrodisiac which one of our previous president demanded on Sri-Lankan airline flights.

I prohibit this delicacy to presidents and prime ministers.

Bottom line is, the temporary loss of memory is an early sign of Dementia and that is the time for one to act and not later.

This transient memory loss comes after 30 years and certainly after 45 years and is probably related to prolonging (lag) of late cell cycles in a strict 50 Cell Cycles events in biology.

Astroglia has some role in this.

To learn about the cell cycle read my book on three demented dogs, coming after the Ceylon Elections. This is the summary of the 1st chapter on Three Dogs with Dementia funded by none other than Maha Brahma, himself.

Humour (not political satire) is the best Medicine and this is what we lack during our extended Election Campaign.

By the way, I sleep well in my retirement.

There is some relationship (brain activity) of calcium metabolism which I do not understand in memory channels or networks. I think vitamin D has some relationship which we do not lack in Ceylon (probably early morning hours, later in the day there are more UV light damage). In the West taking Vitamin D supplement is mandatory. There is special Vitamin supplement for those who are over 50.

Vitamin B1 and B6 are vital for brain function and Hemoglobin level is also important. In my case,      I have too much of it from early days of my life probably related to sports activity. 

I do not think I have an understanding renal tumour.